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Uriprim (Allopurinol)

Uriprim is used for treating gout caused by excessive levels of uric acid in the blood (hyperuricemia). It is a xanthine oxidase inhibitor and is similar to febuxostat (Uloric). Uric acid is a by product from the breakdown of certain proteins (purines) in the body by enzymes called xanthine oxidases. Hyperuricemia occurs when the body produces more uric acid than it can eliminate. The uric acid forms crystals in joints (gouty arthritis) and tissues, causing inflammation and pain. Elevated blood uric acid levels also can cause kidney disease and kidney stones. Uriprim prevents the production of uric acid by blocking the activity of the enzymes that converts purines to uric acid. Uric acid levels usually begin to fall within 2-3 days of starting treatment and return to their original levels within 7-10 days after Uriprim is stopped. It may take several months of therapy before attacks of gout are controlled. The FDA approved Uriprim in August 1966.

Other names for this medication:
Allopurinol, Zyloprim, Allosig, Allupol, Aloc, Alomaron, Alopron, Aluron, Darinol, Goutex, Llanol, Loric, Prodec, Puribel, Puricos, Purinase, Purinol, Talol, Tipuric, Uriprim, Zurim,

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Kifluzol, Sulotrim, Selgene, Perkinil, Biaxsig, Grepid, Supridol, Coversum, Norpilen, Novax, Omep, Ziagen, Noloten, Sedate, Corvo, Flamygel, Panklav, Lucen, Drusa, Cabgolin, Amfebutamone, Carbex, Fenofibrato, Procap, Osiren,

Uriprim is used to treat gout, high levels of uric acid in the body caused by certain cancer medications, and kidney stones. Uriprim is in a class of medications called xanthine oxidase inhibitors. It works by reducing the production of uric acid in the body. High levels of uric acid may cause gout attacks or kidney stones. Uriprim is used to prevent gout attacks, not to treat them once they occur.

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Blepharospasm, oromandibular, lingual, laryngeal and cervical dystonia are common forms of adult-onset dystonia. Tubulolobular carcinoma of the breast: clinical, mammographic and sonographic findings.

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Several mutant forms of PRL-R were constructed either by truncation or by deletion of the cDNA. Single lipopolysaccharide-reactive B cells in the non-immune mouse spleen cell population secrete natural multispecific autoantibodies.

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Filoviruses have the capacity to cause lethal outbreaks of hemorrhagic fever in primates. Medical records were reviewed retrospectively for clinical data. The contribution of epistatic pleiotropy to the genetic architecture of covariation among polygenic traits in mice.

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When realizable physical structure is considered, multi-layer approximation should be applied. Quality of life in Polish respondents: psychometric properties of the Polish WHOQOL-Bref.

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